We have been successful in restoring kids from autism with the use of lasers, dietary changes and supplements. Most kids make significant changes in less than six months.

Manganese is a metal that is needed in small amounts to make enzymes work. However, too much is toxic. Infants need 0.5 mg per day. Adults need 2-4 mg per day.

A problem is that infants cannot excrete excess manganese and thus it is deposited in the basal ganglia of the brain. The basal ganglia are the “traffic director” of the brain. Although the maximum dose allowed for manganese for infants is 0.5 mg, soy infant formula contains about 470 mg per day! In addition, giving a child soy infant formula is like giving them five birth control pills per day. Also, soy inhibits the absorption of iron, making infants iron deficient (remember iron deficiency causes many problems before it causes anemia). Soy also shuts down thyroid function. Soy infant formula has never undergone the required safety tests that the FDA’s own rules require!

Next you must know that two toxic metals is 100 to 1000 times as toxic as one metal. So when we give vaccines, we add more toxic metals. All vaccines contain high levels of tin and aluminum. Many, including all flu vaccines, contain mercury as well. Most dental fillings put in kids also contain mercury. Thus we have a mix of manganese, tin, aluminum, and mercury. Most commonly, autistic kids will have been given soy infant formula and then become autistic soon after their 18-month vaccinations.


The person responsible for calling attention to manganese in autism is Shauna Young, CTN

"Manganese (Mn) status was determined in 24 infants exclusively fed either human milk (n = 8) or formula (n = 16) from birth to 3 months. Infant formulas were observed to contain considerably higher levels of Mn (70.0 to 1289.0 micrograms/L) than either human milk (means = 4.9 micrograms/L) or cows' milk (means 25.2 micrograms/L). At 3 months, human milk-fed infants consumed a smaller volume of milk (689.0 ml) than formula-fed infants (894.0 ml), and also received significantly less Mn (0.42 micrograms/kg/day) than formula-fed infants (183.22 micrograms/kg/day)."

Am J Clin Nutr (1984 Jun) 39(6): 872-8

That means that most formula-fed infants are getting from 2-4 mg of manganese per day with any over 0.5 mg being toxic.

"Infant formula, ABBOTT NUTRITION, NEOSURE ADVANCE, powder, with ARA and DHA, (formerly ROSS) contains 51 mg of manganese per 100 grams."

This is 15.7 mg manganese per ounce.

Soy infant formulas are about 400% higher in manganese than breast milk! This means that a child getting 30 ounces of this formula would consume about 471 mg of manganese per day!

3/4 cup of Cheerios has 0.8 mg of manganese = over the allowable for an infant!

You can Google "manganese madness" or "manganese toxicity" and see that the damaging effects of manganese on the nervous system is well documented.

Effect of high dietary manganese intake of neonatal rats on tissue mineral accumulation, striatal dopamine levels, and neurodevelopmental status.

Tran TT,Chowanadisai W,Crinella FM,Chicz-DeMet A,Lonnerdal B

Neurotoxicology (2002 Oct) 23(4-5): 635-43ISSN: 0161-813X

Abstract: Mn is an essential element, but may become neurotoxic at high levels. Recent reports of high Mn levels in hair of children with neurodevelopmental deficits suggest that these deficits could be due to Mn-induced neurotoxic effects on brain dopamine (DA) systems, although the mechanism is not well understood. Infant formulas contain considerably higher concentrations of Mn than human milk. Thus, formula-fed infants are exposed to high levels of Mn at a time when Mn homeostasis is incompletely developed. We studied the effects of dietary Mn supplementation of rat pups on tissue Mn accumulation, brain dopamine levels, infant neurodevelopmental status, and behavior at maturity. Newborn rats were supplemented daily with 0, 50, 250, or 500 microg Mn given orally from day 1 to day 20. Mineral analysis of small intestine and brain at day 14 showed a significant increase of tissue Mn in supplemented rats. Neurodevelopmental tests conducted at various ages showed significant delays as a function of Mn supplementation. At day 32, there was a significant positive relationship between passive avoidance errors and Mn supplementation levels. Brains of animals killed on day 40 showed a significant inverse relationship between Mn supplementation level and striatal dopamine concentration. These observations suggest that dietary exposure to high levels of Mn during infancy can be neurotoxic to rat pups and result in developmental deficits.